By Martin Stefan
This entire colour atlas covers the scientific gains, etiology, pathophysiology, and remedy of schizophrenia together with smooth advancements in therapy and instructions of latest learn. It describes schizophrenia's profound results on belief, realizing, communique, and social habit; its identifiable antecedents in early mind development and improvement; and morphological information from structural mind imaging and autopsy reports. The atlas offers examples of the newest study advancements in neuropsychology, psychophysiology and useful imaging besides in vivo receptor imaging and different advances in psychopharmacology.
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Extra info for An Atlas of Schizophrenia
Figure reproduced with permission from Laruelle M, Abi-Dargham A, Gil R, et al. Increased dopamine transmission in schizophrenia: relationship to illness phases [Review]. Biol Psychiatry 1999; 46:56–72 CLASSIFICATION OF DOPAMINE RECEPTORS Dopamine D2 receptor family D1 receptor family D1 D5 D2 D3 D4 Caudateputamen N. accumbens Olfactory tubercule Hippocampus Hypothalamus Caudateputamen N. accumbens Olfactory tubercule Olfactory tubercule Hypothalamus N. 5 There are currently five types of dopamine receptor identified in the human nervous system, D1 to D5.
Evidence concerning cannabis comes from the Swedish army study19 in which army recruits were interviewed about their drug consumption and then followed-up for a decade and a half. 12). 13 The rate of life events is increased in schizophrenia, although the effect is not as great as in depression. Figure reproduced with permission from Bebbington P, Wilkins S, Jones P, et al. Life events and psychosis. Initial results from the Camberwell Collaborative Psychosis Study. Br J Psychiatry 1993;162:72–9 FREQUENCY OF LIFE EVENTS Life event rates per100 subjects 100 Schizophrenia Mania Depressive psychosis Controls 90 80 70 60 50 40 30 20 10 0 1 2 3 4 5 6 Months before onset/interview It is estimated that 20–50% of the population with schizophrenia in Western countries may qualify as substance abusers or ‘dual-diagnosis’ patients.
E. we do not understand how the causes ‘cause’ schizophrenia. 4 Causality over the life course. 5 Lifetime risk of developing schizophrenia in relatives of schizophrenic individuals. 6 Concordance rates for schizophrenia in studies of monozygotic and dizygotic twins. The implication of the lack of 100% concordance in monozygotic twins is that there must be environmental etiological factors involved in the genesis of schizophrenia. 1 Lifetime expectancy of broadly defined schizophrenia in the relatives of schizophrenics.
An Atlas of Schizophrenia by Martin Stefan