By Karl Maramorosch
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Extra info for Advances in Virus Research, Volume 71
2004). For the most part, the results were similar to those reported by Ren and Racaniello (1992a,b). , 2004), closely resembling the behavior of the MV strain in monkeys (Flexner, 1931; Howe and Bodian, 1942). , 2002). In these mice, poliovirus replicated mainly in glial and ependymal cells in the CNS and produced a pathological picture totally different from poliomyelitis. , 2005). These mice could be infected by mouth with a large virus dose that produced paralysis in 50% of the animals. Poliovirus replicated to high titer in many visceral organs and there was a high titer viremia.
And Howe, C. L. (2005). Picornaviruses and cell death. Trends Microbiol. 14:28–36. , and Carrasco, L. (2004). Individual expression of poliovirus 2Apro and 3Cpro induces activation of caspase-3 and PARP cleavage in HeLa cells. Virus Res. 104:39–49. , and O’Brien, S. J. (2003). The influence of HLA genotype on AIDS. Annu. Rev. Med. 54:535–551. , and Pelletier, I. (1989). Persistent poliovirus infection of human neuroblastoma cells. Proc. Natl. Acad. Sci. 86:7590–7594. , Gosselin, A. , and Blondel, B.
And Green, H. (1973). Picornavirus receptors and picornavirus multiplication in human mouse hybrid cell lines. Virology 54:515–524. Melnick, J. L. (1952). Latent survival of viruses and persistence of immunity. In ‘‘Poliomyelitis: Papers and Discussions Presented at the Second International Poliomyelitis Conference,’’ pp. 301–318. B. Lippincott, Philadelphia. , Lionetti, K. , and Racaniello, V. R. (1986). Transformation of a human poliovirus receptor gene into mouse cells. Proc. Natl. Acad. Sci.
Advances in Virus Research, Volume 71 by Karl Maramorosch